| [1] Abe K，Kimura H.The possible role of hydrogen sulfide as an endogenous neuromodulator[J].J Neurosci, 1996, 16(3): 1066-1071.[2] Zhao WM，Zhang J，Lu YJ，et al.The vasorelaxant effect of H2S as a novel endogenous gaseous K-ATP channel opener[J].Embo J, 2001, 20(21): 6008-6016.[3] Yang G，Sun X，Wang R.Hydrogen sulfide-induced apoptosis of human aorta smooth muscle cells via the activation of mitogen-activated protein kinases and caspase-3[J].FASEB J, 2004, 18(14): 1782-1784.[4] Patel P，Vatish M，Heptinstall J，et al.The endogenous production of hydrogen sulphide in intrauterine tissues[J].Reprod Biol Endocrinol, 2009, 7(10)：doi: 10.1186/1477-7827-7-10.[5] American College of Obstetricians and Gynecologists.ACOG Practice Bulletin. Assessment of risk factors for preterm birth. Clinical management guidelines for obstetrician-gynecologists. Number 31, October 2001. (Replaces Technical Bulletin number 206, June 1995; Committee Opinion number 172, May 1996; Committee Opinion number 187, September 1997; Committee Opinion number 198, February 1998; and Committee Opinion number 251, January 2001)[J].Obstet Gynecol, 2001, 98(4): 709-716.[6] Zhang L，Sio SWS，Moochhala S，et al.Role of hydrogen sulfide in severe burn injury-induced inflammation in mice[J].Mol Med, 2010, 16(9-10): 417-424.[7] Fiorucci S，Orlandi S，Mencarelli A，et al.Enhanced activity of a hydrogen sulphide-releasing derivative of mesalamine (ATB-429) in a mouse model of colitis[J].Brit J Pharmacol, 2007, 150(8): 996-1002.[8] Li L，Bhatia M，Zhu YZ，et al.Hydrogen sulfide is a novel mediator of lipopolysaccharide-induced inflammation in the mouse[J]. FASEB J, 2005, 19(6): 1196-1205.[9] Sidhu R，Singh M，Samir G，et al.L-cysteine and sodium hydrosulphide inhibit spontaneous contractility in isolated pregnant rat uterine strips in vitro[J].Pharmacol Toxicol, 2001, 88(4): 198-203.[10] You XJ，Xu C，Lu JQ，et al.Expression of cystathionine β-synthase and cystathionine gamma-lyase in human pregnant myometrium and their roles in the control of uterine contractility[J].Plos One, 2011, 6(8): doi: 10.1371/journal.pone.0023788.[11] Zhao P，Huang X，Wang ZY，et al.Dual effect of exogenous hydrogen sulfide on the spontaneous contraction of gastric smooth muscle in guinea-pig[J].Eur J Pharmacol, 2009, 616(1-3): 223-228.[12] Hosoki R，Matsuki N，Kimura H.The possible role of hydrogen sulfide as an endogenous smooth muscle relaxant in synergy with nitric oxide[J].Biochem Bioph Res Co, 1997, 237(3): 527-531.[13] Li L，Whiteman M，Guan YY，et al.Characterization of a novel, water-soluble hydrogen sulfide - releasing molecule (GYY4137):unew insights into the biology of hydrogen sulfide[J].Circulation, 2008, 117(18): 2351-2360.[14] Maynard SE，Min JY，Merchan J，et al.Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfimction, hypertension, and proteinuria in preeclampsia[J].J Clin Invest, 2003, 111(5): 649-658.[15] Venkatesha S，Toporsian M，Lam C，et al.Soluble endoglin contributes to the pathogenesis of preeclampsia (vol 12, pg 642, 2006)[J].Nat Med, 2006, 12(7): 862-873.[16] Wang KQ，Ahmad S，Cai M，et al.Dysregulation of hydrogen sulfide producing enzyme cystathionine gamma-lyase contributes to maternal hypertension and placental abnormalities in preeclampsia[J].Circulation, 2013, 127(25): 2514-2522.[17] Li L，Hsu A，Moore PK.Actions and interactions of nitric oxide, carbon monoxide and hydrogen sulphide in the cardiovascular system and in inflammation - a tale of three gases![J].Pharmacol Therapeut, 2009, 123(3): 386-400.[18] Cindrova-Davies T，Herrera EA，Niu YG，et al.Reduced cystathionine gamma-lyase and increased mir-21 expression are associated with increased vascular resistance in growth-restricted pregnancies hydrogen sulfide as a placental vasodilator[J].Am J Pathol, 2013, 182(4): 1448-1458.[19] Xu S, Liu Z, Liu P.Targeting hydrogen sulfide as a promising therapeutic strategy for atherosclerosis[J].Int J Cardiol，2014，172(2):313-317. （2014-02-01收稿 2014-05-29修回） |
